Abstract
Introduction. Coronary slow flow (CSF) phenomenon, also known as cardiac syndrome Y, is defined as the delayed coronary opacification. Different hypotheses have been postulated about its mechanism, such as inflammation and endothelial dysfunction. Several studies have confirmed that the inflammatory response is also important in the pathogenesis of tuberculosis. Materials and methods. Retrospective study. We investigated the effect of tuberculosis inflammation on the frequency of the CSF in 83 patients with tuberculosis (investigation group) and 560 patients without tuberculosis (control group). Results. We found that CSF has an incidence of 6% in control group, and 23% in patients with tuberculosis. In both groups, it was functional for some patients (corrected by nitrates), and fixed for others. Further studies are still necessary.